rabbit anti rat nf κb p65 immunoglobulin g (Cell Signaling Technology Inc)
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rabbit anti rat nf κb p65 immunoglobulin g
Rabbit Anti Rat Nf κb P65 Immunoglobulin G, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/g+anti+p65/pm39734479-148-1-7
Average 86 stars, based on 1 article reviews
Rabbit Anti Rat Nf κb P65 Immunoglobulin G, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/g+anti+p65/pm39734479-148-1-7
Average 86 stars, based on 1 article reviews
rabbit anti rat nf κb p65 immunoglobulin g - by Bioz Stars,
2026-09
86/100 stars
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Sonication:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Shear:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Immunoprecipitation:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Activation Assay:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Expressing:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. RNA Sequencing Assay:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Plasmid Preparation:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Transfection:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Western Blot:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Immunofluorescence:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Positive Control:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Staining:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Stable Transfection:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Luciferase:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Activity Assay:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Comparison:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Quantitative RT-PCR:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Incubation:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Mutagenesis:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Construct:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Translocation Assay:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. Migration:Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway Article Snippet: The cell lysates were sonicated to shear DNA to sizes of 200–500 bp and centrifuged at 12,000 g at 4 °C for 10 min. To preclear the chromatin, 60 μL of protein G agarose was added to each tube before immunoprecipitated with 5 μ g Article Title: DHX9 contributes to the malignant phenotypes of colorectal cancer via activating NF-κB signaling pathway. Article Snippet: Colorectal cancer (CRC) is the leading cause of cancer-related mortality worldwide, which makes it urgent to identify novel therapeutic targets for CRC treatment.. In this study, DHX9 was filtered out as the prominent proliferation promoters of CRC by siRNA screening.. Moreover, DHX9 was overexpressed in CRC cell lines, clinical CRC tissues and colitis-associated colorectal cancer (CAC) mouse model. |